Abstract
The infection by the human immunodeficiency virus has spread to all corners of the planet, being one of the deadliest infections of the last century. In this population, several cardiovascular manifestations have been identified, including heart failure. In general, it is a late complication of human immunodeficiency virus infection. Multiple pathophysiological mechanisms involved in metabolic and cardiovascular disorders that can cause long-term heart failure have been described. Among the clinical manifestations in patients with human immunodeficiency virus, the signs of congestive heart failure have been detected: dyspnea, fatigue, edema, among others. Currently, strategies to reduce the risk of heart failure include the early establishment of antiretroviral therapy with minimal possible metabolic effects and careful treatment of traditional cardiovascular risk factors.
Keywords
References
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